Cart is empty
This product can be shipped into all countries.
Folic Acid (vitamin B9) is primarily indicated for folate deficiencies and during pregnancy to prevent foetal spinal deformities. It also reduces the body’s homocysteine levels. Elevated homocysteine is associated with cardiovascular disease. However, research is unclear whether homocysteine is a cause of cardiovascular disease, or if it is a marker (effect) of existing cardiovascular disease. Research published in the medical journal Stroke, indicates that folic acid may reduce the risk of strokes, an effect which may be independent of its effect on homocysteine. Folic acid may also help relieve depression and boost cognitive function.SOLAL Folic Acid contains the natural form of folic acid, not the synthetic form found in most supplements.
Folate (vitamin B9) is primarily indicated for folate deficiencies and during pregnancy to prevent foetal spinal deformities. Folate is involved in the metabolism of homocysteine. Low folate levels are associated with increased plasma homocysteine levels. High homocysteine may be a risk factor for certain chronic diseases. Folate deficiency leads to the formation of abnormal red blood cells called megaloblasts. DEPLETIONS The following medicines and substances can cause a folate deficiency. If you take any of these then it is particularly important that you supplement with folate under medical supervision: alcohol, antacids, antibiotics, anti-inflammatories, carbamazepine, diuretics, estrogens, metformin, methotrexate, methylprednisolone, phenobarbital, phenytoin, sulfasalazine, triamterene, trimethoprim and valproic acid.
Each tablet contains the following as active ingredient:
Folate (L-5-methyltetrahydrofolate calcium) 750µg
Store in a cool dry place below 25°C. Keep, out of the reach of children.
Folate is the natural form of folic acid found in food. Once absorbed folate enters the methylation cycle. In humans, tetrahydrofolate- based enzymes play a major role in intracellular metabolism. Tetrahydrofolate plays an indirect role in the rate-limiting step of DNA synthesis. Abnormalities in this process that occur with folate deficiency can cause megaloblastic anaemia. Folate is also involved in the metabolism of homocysteine. Low folate levels are associated with increased plasma homocysteine levels. Remethylation of homocysteine to methionine requires folic acid and vitamin B12 as cofactors. Folate supplementation has been shown to reduce the risk of neural tube defects in the foetus.